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The staff additionally used gene therapy to reactivate the chaperone protein in a mouse mannequin and [Memory Wave](https://wiki.drawnet.net/index.php?title=Usu%C3%A1rio:RoscoeDexter) found that the memory deficit was reversed, confirming that the protein folding equipment acts as a molecular change for memory. "Identifying this protein folding mechanism is an important step towards understanding how memories are stored and what goes unsuitable in diseases associated with memory impairment," Abel says. In addition to Chatterjee and Abel, the research team included Jacob Michaelson, UI associate professor of psychiatry \ No newline at end of file